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Aortic stenosis (AS) augments total LV afterload and elevates E a /E es. In this setting, E a reflects the combination of arterial and valvular resistances and is generally markedly increased. Pressure rises sharply during systole to a domed-shaped PV loop.
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This review will focus on clinical manifestations and diagnosis of LVNC as an isolated disorder distinct from other clinical settings in which non-compacted myocardium may be seen in association with other cardiac and noncardiac abnormalities. Management and prognosis of isolated LVNC is discussed separately.
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